|optic nerve injury
|siRNA injection, in situ hybridization, qRT-PCR, Western blot, immunohistochemical analysis
|RGC apoptosis induced by optic nerve injury involves activation of caspase-2, and that synthetic siRNAs designed to inhibit expression of caspase-2 represent potential neuroprotective agents for intervention in human diseases involving RGC loss.
|Ocular neuroprotection by siRNA targeting caspase-2.
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